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Initially, the TlpB receptor on host cells is triggered by chemical signals, promoting a chemotactic response toward the gastric epithelium ( Helicobacter pylori navigates through the acidic environment via its flagella, avoiding direct gastric acid-induced damage, and after traversing the relatively neutral mucus layer, it firmly adheres to gastric epithelial cells ( Helicobacter pylori secretes urease, which hydrolyzes urea to produce ammonia and carbon dioxide, locally neutralizing gastric acid and creating a microenvironment favorable for its survival ( H

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Cancers Achilles heel: apoptosis and necroptosis to the rescue