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Genetic elimination of K ATP channels ( Kir6.2 / mice) significantly increases glucose absorption and glucose-stimulated GIP secretion, through a compensatory increase in duodenal Sglt1 mRNA expression (Ogata et al., 2014)

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By directing the delivery of the NMDA receptor antagonist MK-801 via GLP-1 pathways, the researchers were able to influence neuroplasticity in the hypothalamus and brainstem