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Metabolic parameters (serum glucose and insulin, lipid profile, urinary albumin, and creatinine levels) showed no significant differences before and after treatment

Complementing these disease-specific examples, epigenome-wide studies in type 1 diabetes (T1D) have demonstrated methylation abnormalities in immune effector cells (CD4+ T cells, B cells and monocytes), underscoring that immune epigenomes are broadly sensitive to both inflammatory context and metabolic state (112,113)

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AOD 9604 functions by targeting specific receptors involved in fat metabolism, particularly those that trigger the breakdown of fats into fatty acids